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Jian Kuirong



Jiang Kuirong is a chief physician, professor and PHD supervisor. He currently works as the member of Operation Group of Surgery Branch of Chinese Medical Association, a youth committee member of Surgery Branch of Chinese Medical Association, the member of Surgical Oncology Physicians Committee of Chinese Medical Association, Fellow of American College of Surgeons (FACS), Fellow of International College of Surgeons (FICS), Deputy Chief of Pancreas Surgery Group of Surgery Branch of Jiangsu Province Medical Association, the member of Surgery Branch of Jiangsu Province Medical Association, the member of Surgery Branch of Jiangsu Province Physicians Association, the member and Secretary of Pancreas Disease Branch of Jiangsu Province Medical Association, Chairman of Jiangsu Province Pancreatic Disease Society, the member of General Surgery Specialty Committee of Chinese Research Hospital Association, the member of Hepatopancrea-tobiliary Surgery ERAS Specialty Committee of Chinese Research Hospital Association, and the member of Minimally Invasive Diagnosis and Treatment Group of Pancreatic Cancer Specialty Committee of Chinese Anti-Cancer Association.

Prof. Jiang is now the high-level personnel cultivation object of "Six-industry Talent Summit" of Jiangsu Province, the cultivation object of Jiangsu Province "333" Project, the young academic leader of "Qinglan Project" of Jiangsu Colleges and Universities, outstanding young and middle-aged academic leader of "Outstanding Young and Middle-aged Teacher Support Plan" of Nanjing Medical University.

Participated in the “Innovation and Application of Key Medical Technologies of Pancreatic Cancer” project as a main contributor, and won the First Prize of 2015 Jiangsu Provincial Prize for Science and Technology (ranked the third); “The Study of the Foundation and New Clinical Technologies of Pancreatic Cancer” won the First Prize of 2015 Jiangsu Provincial Award for Medical Science and Technology (ranked the third); “The Occurrence Mechanism and Development Mechanism and Clinical Technological Innovation Study of Pancreatic Cancer” won the Second Prize of 2015 China Award for Medical Scientific and Technological Progress (ranked the third); “The Middle Segment Pancreatectomy” won the First Prize of 2013 Jiangsu Provincial Health Department Award for New Medical Technology Introduction (ranked the second); “The Foundation Study of Clinical Study of Pancreatic Cancer” won the Second Prize of 2012 Ministry of Education Award for Scientific and Technological Progress (ranked the third).



Personnel programs and scientific research tasks involved

2013.1-2016.12. The person in charge of the Study of the Mechanism of How the High Expression Galectin-1 of Pancreatic Stellate Cell Guides the Hepatic Metastasis in Pancreatic Cancer via SDF-1/CXCR4 Biological Axis, Natural Science Foundation of China

2010.1-2012.12. The person in charge of the Study of the Mechanism of How the High Expression Galectin-1 of Pancreatic Stellate Cell Promotes Pancreatic Cancer Immunosuppression, Natural Science Foundation of China

2013.1-2017.1. The main contributor of the subsidiary subject of the Translational Medicine Study of the Early Diagnosis and Comprehensive Treatment of Pancreatic Cancer, the Industry Fund of the Ministry of Health

2009.1-2013.1. The main contributor of the subsidiary subject of The Systematic Study of the Comprehensive Therapeutic System of Pancreatic Cancer, the “11th Five-Year Plan” Support Program of the Ministry of Science and Technology

2014.11-2018.12. The person in charge of the Study of the Mechanism of How Pancreatic Stellate Cell and the High Expression HIF-1α of Pancreatic Cancer Cell Regulate Tumor Invasion and Metastasis, the High-Level Talented Personnel Training Program (Type B Program) of Jiangsu’s “Six-industry Talent Summit”


Reference

1.    WuJ, Guo F, Wei J, et al. [Surgical treatment for pancreatic neuroendocrineneoplasmas]. Zhejiang da xue xue bao Yi xue ban = Journal of Zhejiang UniversityMedical sciences 2016; 45(1): 31-5.

2.    WeiJ, Liu X, Wu J, et al. Diagnosis and surgical management of insulinomas in 33consecutive patients at a single institution. Langenbeck's archives of surgery2016; 401(7): 1019-25.

3.    TuM, Lu C, Lv N, et al. Vasohibin 2 promotes human luminal breast cancerangiogenesis in a non-paracrine manner via transcriptional activation offibroblast growth factor 2. Cancer letters 2016; 383(2): 272-81.

4.    QianD, Lu Z, Jackson R, et al. Effect of antecolic or retrocolic route ofgastroenteric anastomosis on delayed gastric emptying afterpancreaticoduodenectomy: A meta-analysis of randomized controlled trials.Pancreatology : official journal of the International Association ofPancreatology (IAP)  [et al] 2016; 16(1):142-50.

5.    LuZ, Yin J, Wei J, et al. Small amounts of tissue preserve pancreatic function:Long-term follow-up study of middle-segment preserving pancreatectomy. Medicine2016; 95(46): e5274.

6.    HuoX, Wei J, Liu X, et al. Brunner's gland cyst in combination withgastrointestinal stromal tumor: A case report. Oncology letters 2016; 11(5):3409-12.

7.    GaoW, Dai X, Dai C, et al. Comparison of patency rates and clinical impact ofdifferent reconstruction methods following portal/superior mesenteric vein resectionduring pancreatectomy. Pancreatology : official journal of the InternationalAssociation of Pancreatology (IAP)  [etal] 2016; 16(6): 1113-23.

8.    ZouC, Lu Z, Zhang Z, et al. Factors associated with pancreatic infection inpatients with severe acute pancreatisis. International journal of clinical andexperimental medicine 2015; 8(8): 14100-4.

9.    WuQ, Zhang M, Qin Y, et al. Systematic review and meta-analysis of isletautotransplantation after total pancreatectomy in chronic pancreatitispatients. Endocrine journal 2015; 62(3): 227-34.

10.  WeiJ, Liu X, Wu J, et al. Modified One-layer Duct-to-mucosa PancreaticojejunostomyReduces Pancreatic Fistula After Pancreaticoduodenectomy. International surgery2015.

11.  WeiJ, Lin S, Wang C, et al. Glucagonoma syndrome: A case report. Oncology letters2015; 10(2): 1113-6.

12.  TangD, Gao J, Wang S, et al. Apoptosis and anergy of T cell induced by pancreaticstellate cells-derived galectin-1 in pancreatic cancer. Tumour biology : thejournal of the International Society for Oncodevelopmental Biology and Medicine2015; 36(7): 5617-26.

13.  GeQ, Zhou J, Tu M, et al. Nuclear vasohibin-2 promotes cell proliferation byinducing G0/G1 to S phase progression. Oncology reports 2015; 34(3): 1327-36.

14.  TuM, Liu X, Han B, et al. Vasohibin2 promotes proliferation in human breastcancer cells via upregulation of fibroblast growth factor2 andgrowth/differentiation factor15 expression. Molecular medicine reports 2014;10(2): 663-9.

15.  TangD, Zhang J, Yuan Z, et al. Pancreatic satellite cells derived galectin-1increase the progression and less survival of pancreatic ductal adenocarcinoma.PloS one 2014; 9(3): e90476.

16.  TangD, Wang D, Yuan Z, et al. Persistent activation of pancreatic stellate cellscreates a microenvironment favorable for the malignant behavior of pancreaticductal adenocarcinoma. International journal of cancer 2013; 132(5): 993-1003.

17.  SunJ, Tu M, Han B, et al. Generation and characterization of rabbit polyclonalantibodies against Vasohibin-2 for determination of its intracellularlocalization. International journal of oncology 2013; 43(1): 255-61.

18.  ChenJ, Li Q, An Y, et al. CEACAM6 induces epithelial-mesenchymal transition andmediates invasion and metastasis in pancreatic cancer. International journal ofoncology 2013; 43(3): 877-85.

19.  CaiB, An Y, Lv N, et al. miRNA-181b increases the sensitivity of pancreatic ductaladenocarcinoma cells to gemcitabine in vitro and in nude mice by targetingBCL-2. Oncology reports 2013; 29(5): 1769-76.

20.  AnY, Cai B, Chen J, et al. MAP3K10 promotes the proliferation and decreases thesensitivity of pancreatic cancer cells to gemcitabine by upregulating Gli-1 andGli-2. Cancer letters 2013; 329(2): 228-35.

21.  TangD, Yuan Z, Xue X, et al. High expression of Galectin-1 in pancreatic stellatecells plays a role in the development and maintenance of an immunosuppressivemicroenvironment in pancreatic cancer. International journal of cancer 2012;130(10): 2337-48.

22.  XueX, Lu Z, Tang D, et al. Galectin-1 secreted by activated stellate cells inpancreatic ductal adenocarcinoma stroma promotes proliferation and invasion ofpancreatic cancer cells: an in vitro study on the microenvironment ofpancreatic ductal adenocarcinoma. Pancreas 2011; 40(6): 832-9.